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Western Diseases (western + disease)
Selected AbstractsBook review: Western Diseases: An Evolutionary PerspectiveAMERICAN JOURNAL OF HUMAN BIOLOGY, Issue 2 2009Peter D. GluckmanArticle first published online: 6 JAN 200 No abstract is available for this article. [source] Book review: Western Diseases: An Evolutionary PerspectiveAMERICAN JOURNAL OF PHYSICAL ANTHROPOLOGY, Issue 4 2009Kenneth M. Weiss No abstract is available for this article. [source] An Eastern art form for a Western disease: randomised controlled trial of yoga in patients with poorly controlled insulin-treated diabetesPRACTICAL DIABETES INTERNATIONAL (INCORPORATING CARDIABETES), Issue 6 2002Dr David Kerr MD FRCP FRCPE Consultant Physician Abstract There is increasing interest in the use of complimentary therapies. At present, there is a paucity of data examining the effectiveness of such therapies in patients with diabetes. We have examined the influence of providing yoga for patients with diabetes that is poorly controlled despite the best efforts of the multidisciplinary team. After obtaining ethical approval, 37 patients with poorly controlled diabetes were randomised to a traditional intensive education programme and simple exercises or a 16 week (32 session) Hatha yoga plan. Participation in regular yoga sessions did not improve glycaemic control but insulin requirements remained stable in the yoga group and increased in the controls. Although quality of life was not altered, all but one subject in the yoga group opted to continue with yoga long term after completion of the study. Copyright © 2002 John Wiley & Sons, Ltd. [source] Milk consumption: aggravating factor of acne and promoter of chronic diseases of Western societiesJOURNAL DER DEUTSCHEN DERMATOLOGISCHEN GESELLSCHAFT, Issue 4 2009Bodo Melnik Summary Consumption of cow's milk and cow's milk protein result in changes of the hormonal axis of insulin, growth hormone and insulin-like growth factor-1(IGF-1) in humans. Milk consumption raises IGF-1 serum levels in the perinatal period, adolescence and adulthood. During puberty with the physiological onset of increased secretion of growth hormone, IGF-1 serum levels increase and are further enhanced by milk consumption. IGF-1 is a potent mitogen; after binding to its receptor in various tissues, it induces cell proliferation and inhibits apoptosis. Keratinocytes and sebocytes, as well as the androgen-synthesizing adrenals and gonads, are stimulated by IGF-1. The epidemic incidence of adolescent acne in Western milk-consuming societies can be explained by the increased insulin- and IGF-1-stimulation of sebaceous glands mediated by milk consumption. Acne can be regarded as a model for chronic Western diseases with pathologically increased IGF-1-stimulation. Many other organs, such as the thymus, bones, all glands, and vascular smooth muscle cells as well as neurons are subject to this abnormally increased hormonal stimulation. The milk-induced change of the IGF-1-axis most likely contributes to the development of fetal macrosomia, induction of atopy, accelerated linear growth, atherosclerosis, carcinogenesis and neurodegenerative diseases. Observations of molecular biology are supported by epidemiologic data and unmask milk consumption as a promoter of chronic diseases of Western societies. [source] | |||||||||||||