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Superior Vena Cava Syndrome (superior + vena_cava_syndrome)

Distribution by Scientific Domains

Medical Sciences	100%

Selected Abstracts

Doppler Superior Vena Cava Flow Evolution and Respiratory Variation in Superior Vena Cava Syndrome

ECHOCARDIOGRAPHY, Issue 4 2008
Fa Qin Lv M.D.
Background: Superior vena cava syndrome (SVCS) is a clinical expression of obstruction of blood flow through the superior vena cava. The patterns of the Doppler flow changes of superior vena cava (SVC), especially the respiratory effects on them have not yet been fully elucidated. This study was to examine SVC Doppler flow patterns and the respiratory effects on them in healthy subjects and patients with SVCS. Methods: The SVC Doppler flow patterns of 18 normal human subjects and 22 patients with SVCS were analyzed at initial diagnosis and were followed up every 2 months for at least 11 months. Results: Among the 22 patients, 5 patients with the tumor near the right atrium oppressing the inferior segment of the SVC had clear VR- and AR-waves, while in the other 17 patients the VR- and AR-waves disappeared or their outlines were vague. The respiratory variations of the S- and D-waves as a percentage change in inspiration compared to expiration in patient group were much lower than those in control group (S-wave: 1.67 ± 3.32% vs. 15.65 ± 16.15%, P = 0.0003; D-wave: 1.80 ± 1.12% vs. 23.55 ± 37%, P = 0.0087), which gradually became larger with treatment and showed no significant difference with those in control group after 7 months. Conclusions: The Doppler flows of the patients with SVCS correlate well with the images of CT scan of them. The respiratory variation of the S- and D-velocities could be used to evaluate the severity of SVC obstruction and its therapeutic effect. [source]

Fibrosing TB Mediastinitis Presenting as a Superior Vena Cava Syndrome: A Case Presentation and Echocardiogram Correlate

ECHOCARDIOGRAPHY, Issue 7 2006
Lidiette Esquivel M.D.
A 49-year-old woman developed a chronic obstruction of the superior vena cava (SVC) as a complication of mediastinal tuberculosis. Echocardiography findings are presented along with the cavography. Symptoms disappeared after stenting the fibrosed SVC. Transesophageal echocardiogram findings led to the condition's final resolution. [source]

Percutaneous Treatment for Pacemaker-Associated Superior Vena Cava Syndrome

PACING AND CLINICAL ELECTROPHYSIOLOGY, Issue 11 2002
ALBERT W. CHAN
CHAN, A.W., et al.:Percutaneous Treatment for Pacemaker-Associated Superior Vena Cava Syndrome. Superior vena cava (SVC) obstruction is an uncommon but serious complication associated with permanent pacemaker implantation. The mechanical stress associated with pacemaker wires may lead to vessel wall inflammation, fibrosis, and thrombus formation, and ultimately to venous stenosis and occlusion. The surgical treatment of pacemaker related SVC syndrome requires thoracotomy and carries significant morbidity. This article illustrates the authors' initial experience with a "one-step" percutaneous approach for this problem, consisting of percutaneous retrieval of a pacemaker system, followed by venous revascularization with angioplasty and stenting, and installation of a new pacemaker device. [source]

Doppler Superior Vena Cava Flow Evolution and Respiratory Variation in Superior Vena Cava Syndrome

Superior vena cava syndrome related to indwelling intravenous catheters in patients with cystic fibrosis

PEDIATRIC PULMONOLOGY, Issue 7 2006
Susan Garwood MD
Abstract Patients with cystic fibrosis (CF) often need long-term implanted vascular-access devices for intravenous antibiotics for chronic lower respiratory tract infections. These devices are not without complications, including infection, occlusion, and vascular thrombosis. Such thrombosis can result in superior vena cava (SVC) syndrome due to the position of the catheter proximal to the right atrium. SVC syndrome in CF patients, however, is rarely reported in the literature, suggesting that its incidence is uncommon. We describe three patients with SVC syndrome as a consequence of implanted vascular-access devices. Pediatr Pulmonol. 2006; 41: 683,687. © 2006 Wiley-Liss, Inc. [source]

Superior vena cava syndrome from an intracardiac non-Hodgkin lymphoma

BRITISH JOURNAL OF HAEMATOLOGY, Issue 5 2006
Ettore Porreca
No abstract is available for this article. [source]

Role of fine-needle aspirate immunophenotyping by flow cytometry in rapid diagnosis of lymphoproliferative disorders

DIAGNOSTIC CYTOPATHOLOGY, Issue 7 2007
Ritu Gupta M.D.
Abstract Immunophenotyping is an essential component in the diagnostic work-up of lymphoproliferative disorders (LPD). As compared to immunohistochemistry, flow cytometric immunophenotyping (FCMI) is rapid, quantitative and a more objective technique. This study was designed to evaluate the utility of FCMI on fine needle aspirates (FNA) in rapid diagnosis of LPD in routine clinical practice. FNA from 31 consecutive cases clinically suggestive of LPD were subjected to FCMI. Representative material for FCMI was obtained in 28 (90%) cases and a definite diagnosis established in 27 cases. Histopathogical correlation was available in 22 cases and concordance with FCMI results was observed in 19 (86.4%) cases. FCMI analysis was inconclusive in 4 cases. The results of FCMI were available the same day and were crucial for therapeutic purpose in 3 patients with superior vena cava syndrome. FCMI combined with cytological examination of aspirate smears permits rapid diagnosis with high level of accuracy resulting in efficient treatment planning for critically ill patients and those from far-off rural areas. Diagn. Cytopathol. 2007;35:381,385. © 2007 Wiley-Liss, Inc. [source]

Pericardial involvement at diagnosis in pediatric Hodgkin lymphoma patients

PEDIATRIC BLOOD & CANCER, Issue 5 2007
Hamid Bashir MD
Abstract Background Because most cases are clinically silent, the incidence, clinical course, and outcome of pericardial involvement in Hodgkin lymphoma are unknown. Methods Records of all patients with newly diagnosed Hodgkin lymphoma treated at our institution between 1991 and 2004 were reviewed. Pericardial involvement was identified by computerized tomography (CT) as focal thickening or nodularity present at the time of diagnosis, and by echocardiography as pericardial effusion. Outcomes measured were incidence of pericardial involvement, relapse-free survival, and overall survival. Results Thirteen of 273 patients (5%) had pericardial involvement. All patients with pericardial involvement had nodular sclerosing tumors versus 183 of 260 patients without pericardial involvement (P,=,0.02); 9 (67%) had a bulky mediastinal mass versus 27% (P,=,0.002). Two patients required pericardial drainage to drain very large effusions (n,=,2). Both patients were symptomatic with either shortness of breath or superior vena cava syndrome. In the 11 cases that did not undergo surgical drainage, the effusion resolved within days after starting chemotherapy. Two patients experienced distant relapse but underwent successful salvage therapy. All 13 patients remain alive and free of disease at a median follow-up of 9.7 years (range, 1.7,12.9 years) with normal cardiac function. Conclusions Pericardial involvement by lymphoma is usually asymptomatic unless accompanied by substantial pericardial effusion. In most cases, pericardial involvement resolves with treatment of the underlying malignancy, but close observation for hemodynamic complications is required. A symptomatic effusion, once treated, does not affect survival. Pediatr Blood Cancer 2007;49:666,671. © 2006 Wiley-Liss, Inc. [source]

Right atrial myeloid sarcoma causing superior vena cava syndrome

BRITISH JOURNAL OF HAEMATOLOGY, Issue 2 2008
Darko Antic
No abstract is available for this article. [source]