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Sulphur Dioxide (sulphur + dioxide)

Distribution by Scientific Domains

Medical Sciences	40%
Business, Economics, Finance and Accounting	13%

Selected Abstracts

Significance of endogenous sulphur-containing gases in the cardiovascular system

CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY, Issue 7 2010
Xin-Bao Wang
Summary 1. The sulphur-containing gases hydrogen sulphide and sulphur dioxide can be generated endogenously in mammalian tissues and exert significant biological effects in the cardiovascular system. Hydrogen sulphide is considered to be the third novel gasotransmitter in addition to nitric oxide and carbon monoxide. The present review describes the effects of hydrogen sulphide on the cardiovascular system and its possible mechanisms under physiological conditions. We also discuss the pathophysiological effects of hydrogen sulphide on cardiovascular diseases. The therapeutic potential of hydrogen sulphide is summarized. 2. We recently discovered that sulphur dioxide, another endogenous sulphur-containing gas, has important physiological and pathophysiological roles in the cardiovascular system. To some extent, the effect of sulphur dioxide is similar to that of the other gasotransmitters nitric oxide, carbon monoxide and hydrogen sulphide. Sulphur dioxide may also be a novel gas mediator in the cardiovascular system. [source]

Profile: air quality regulation policy in Portugal

ENVIRONMENTAL POLICY AND GOVERNANCE, Issue 1 2004
João F. P. Gomes
Ten years after the publication of the first air quality laws in Portugal, and seven years after the publication of the respective emission limits, it seems appropriate to analyse the degree of law enforcement by the Portuguese manufacturing industry. Therefore, based on the emission measurements made regularly by ISQ as the only officially accredited laboratory, a set of about 400 sources is analysed in terms of the compliance with the emission limits regarding total suspended particulate, sulphur dioxide, nitrogen oxides and VOCs. The compliance is evaluated through a non-dimensional parameter and plotted versus the emission flow rate to derive conclusions: it was noticed that emission limits are generally respected regarding sulphur dioxide and nitrogen oxides, which does not happen for the other pollutants considered in this study. However, the non-compliance occurs mainly for very low emission flow rates, which suggests some alterations in the emission limits' applicability, which is being revised at the moment. Copyright © 2004 John Wiley & Sons, Ltd and ERP Environment. [source]

A missing values imputation method for time series data: an efficient method to investigate the health effects of sulphur dioxide levels

ENVIRONMETRICS, Issue 2 2010
Swarna Weerasinghe
Abstract Environmental data contains lengthy records of sequential missing values. Practical problem arose in the analysis of adverse health effects of sulphur dioxide (SO2) levels and asthma hospital admissions for Sydney, Nova Scotia, Canada. Reliable missing values imputation techniques are required to obtain valid estimates of the associations with sparse health outcomes such as asthma hospital admissions. In this paper, a new method that incorporates prediction errors to impute missing values is described using mean daily average sulphur dioxide levels following a stationary time series with a random error. Existing imputation methods failed to incorporate the prediction errors. An optimal method is developed by extending a between forecast method to include prediction errors. Validity and efficacy are demonstrated comparing the performances with the values that do not include prediction errors. The performances of the optimal method are demonstrated by increased validity and accuracy of the , coefficient of the Poisson regression model for the association with asthma hospital admissions. Visual inspection of the imputed values of sulphur dioxide levels with prediction errors demonstrated that the variation is better captured. The method is computationally simple and can be incorporated into the existing statistical software. Copyright © 2009 John Wiley & Sons, Ltd. [source]

Temporal analysis of spatial covariance of SO2 in Europe

ENVIRONMETRICS, Issue 4 2007
Marco Giannitrapani
Abstract In recent years, the number of applications of spatial statistics has enormously increased in environmental and ecological sciences. A typical problem is the sampling of a pollution field, with the common objective of spatial interpolation. In this paper, we present a spatial analysis across time, focusing on sulphur dioxide (SO2) concentrations monitored from 1990 to 2001 at 125 sites across Europe. Four different methods of trend estimation have been used, and comparisons among them are shown. Spherical, Exponential and Gaussian variograms have been fitted to the residuals and compared. Time series analyses of the range, sill and nugget have been undertaken and a suggestion for defining a unique spatial correlation matrix for the overall time period of analysis is proposed. Copyright © 2006 John Wiley & Sons, Ltd. [source]

The meta-analysis of the Italian studies on short-term effects of air pollution (MISA): old and new issues on the interpretation of the statistical evidences

ENVIRONMETRICS, Issue 3 2007
Pierantonio Bellini
Abstract The second meta-analysis of the Italian studies on short-term health effects of air pollution, known as MISA-2, was based on daily time series of indicators of both pollution and of health outcomes. It covered 15 cities during 1996,2002 for a total population of approximately nine millions. Health outcomes included mortality for natural causes, for respiratory diseases and for cardiovascular conditions, as well as hospital admissions for respiratory, cardiac and cerebrovascular diseases. Pollutants considered in univariate analyses were sulphur dioxide (SO2), nitrogen oxide (NO2), carbon monoxide (CO), suspended particulate matter (SPM) measuring less than 10,µm diameter (PM10) and ozone (O3, limited to the summer period). Results, including risk estimates, have been largely confirmatory of findings obtained in previous large meta-analytic studies carried out in North America and in Europe. A full report in Italian is available. The present contribution summarises the results of MISA-2 and addresses three major issues regarding their interpretation: robustness of the causal inferential process, the role of specific air pollutants and the reliability of risk estimates. The former issue is stressed according to Bradford Hill's criteria and the conclusion is reached that at least for the association of air pollution with an increase in mortality the evidence for causality is strong. Assessing the role of each air pollutant is problematic: there is some evidence that the effects of PM10 are partly confounded by other pollutants, but PM10 may not be the best indicator of the role of air SPM (routine measures of PM2.5 have not been introduced in Italy). As for risk estimates, the per cent increase in risk of mortality for unit increase in PM10 concentration, measured in MISA-2, is remarkably similar to estimates in other studies and there is indication for linearity of the dose,response relationship. Copyright © 2007 John Wiley & Sons, Ltd. [source]

Spatial-temporal model for ambient air pollutants in the state of Kuwait

ENVIRONMETRICS, Issue 7 2006
Fahimah A. Al-Awadhi
Abstract In this paper we consider dynamic Bayesian models for four different pollutants: nitric oxide(NO), carbon monoxide(CO), sulphur dioxide(SO2) and non-methane hydrocarbon (NCH4) recorded daily in six different stations in Kuwait from 1999 to 2002. The structure of the models depends on time, space and pollutants dependencies. The approach strives to incorporate the uncertainty of the covariance structure into simulated models and final inference; therefore, hierarchical Bayesian model is applied. Association between level of pollutants and different meteorological variables, such as wind speed, wind directions, temperature and humidity are considered. The models will decompose into two main components: a deterministic part to represent the observed components term and a stochastic term to represent the unobservable components. Our analysis will start with basic model and gradually increase its complexity. At each stage the efficiency of the model will be measured. The resulting models subsequently are tested by comparing the output terms and by comparing and the predictions with the real observations. Copyright © 2006 John Wiley & Sons, Ltd. [source]

Molecular characterization of lactic acid populations associated with wine spoilage

JOURNAL OF BASIC MICROBIOLOGY, Issue 1 2004
L. Beneduce
We have investigated the prevalence of spoilage lactic acid bacteria (LAB) in table wines produced in the Apulia region. The occurrence of LAB was evaluated in wines produced with low sulphur dioxide doses and not supplemented with selected malolactic starters such as Oenococcus oeni. About 150 strains were isolated from wine must and a molecular characterization was performed using PCR-based techniques. Most of the strains analysed belonged to Lactobacillus plantarum species. However, some of the strains were identified as Pediococcus damnosus and Leuconostoc sp. The amplified fragments of Pediococcus damnosus were cloned and sequenced. The coding sequence was highly homologous to that of the ropy plasmid confirming that the isolated strain was a ropy+Pediococcus damnosus. In all the samples analysed, the final must pH value reached was relatively high (from 3.78 to 3.90). The high pH values had probably negatively influenced (counteracted) the activity of sulphur dioxide added, allowing proliferation of spoilage wine microorganisms. [source]

Air pollution and hospitalization for venous thromboembolic disease in Chile

JOURNAL OF THROMBOSIS AND HAEMOSTASIS, Issue 4 2010
R. E. DALES
See also Mannucci PM. Fine particulate: it matters. This issue, pp 659,61; Bonzini M, Tripodi A, Artoni A, Tarantini L, Marinelli B, Bertazzi PA, Apostoli P, Baccarelli A. Effects of inhalable particulate matter on blood coagulation. This issue, pp 662,8. Summary.,Background:,Ambient air pollution is a risk factor for stroke and myocardial infarction, possibly because of alterations in coagulation that influence the arterial circulation. Whether air pollution influences diseases associated with peripheral venous thrombogenesis remains largely unknown. Objectives: To determine the association between air pollution and venous thromboembolic disease (VTE) in a sample of the general population. Methods: A time-series analysis was used to test the association between daily air pollution and VTE hospitalizations in Santiago between 2001 and 2005. Results were adjusted for long-term trends, day of the week and average daily humidex. Results: From a population of 5.4 million, there were, on average, 2.3 admissions for VTE per day. Pooled estimates of relative risk (RR) [95% confidence interval (CI)] of hospitalization for venous disease were: 1.07 (1.05, 1.09) for a 58.4 p.p.b. increase in ozone (O3); 1.06 (1.02, 1.09) for a 5.85 p.p.b. increase in sulphur dioxide (SO2); 1.08 (1.03, 1.12) for a 29.25 ,g/m3 increase in nitrogen dioxide (NO2); and 1.05 (1.03, 1.06) for a 20.02 ,g/m3 increase in particulate matter , 2.5 ,m in mean aerodynamic diameter (PM2.5). For pulmonary embolism (PE) results were: 1.10 (1.07, 1.13) for O3; 1.05 (1.02, 1.08) for SO2; 1.07 (1.04, 1.09) for NO2; and 1.05(1.03, 1.06) for PM2.5, respectively. Conclusion: Air pollution appears to be a risk factor for venous thrombosis and PE, a disease with a significant fatality rate. [source]

Short-term effects of air pollution on cardiovascular diseases: outcomes and mechanisms

JOURNAL OF THROMBOSIS AND HAEMOSTASIS, Issue 11 2007
M. FRANCHINI
Summary. The effects of air pollution on health have been intensively studied in recent years. Acute exposure to environmental pollutants such as particulate and gaseous matters (carbon monoxide, nitrogen oxides, sulphur dioxide and ozone) was associated with an increased rate of events and mortality because of cardiovascular diseases. These effects were investigated in short-term studies, which related day-to-day variations in air pollution to disease, and in long-term studies, which have followed cohorts of exposed individuals over time. The evidence from the literature on the short-term cardiovascular effects of air pollutants is discussed from clinical and mechanistic points of view. [source]

Total serum IgE levels are associated with ambient ozone concentration in asthmatic adults

ALLERGY, Issue 1 2009
E. Rage
Background:, Effects of air pollution exposure on IgE-mediated response in asthmatics are poorly investigated. The aim was to examine the relationship between air pollution concentrations and total IgE levels in adult asthmatics. Methods:, The present study relates to the 369 asthmatic adults from the French Epidemiological study on Genetics and Environment of Asthma (EGEA), with availability of data on both total serum IgE measurements and air pollution concentrations. Geo-statistical models were performed on 4 × 4 km grids to assess individual outdoor air pollution exposure. Annual outdoor concentrations of ozone (O3), nitrogen dioxide (NO2), sulphur dioxide (SO2), and particulate matter smaller than 10 ,m size (PM10), and concentrations of summer ozone were assigned to subject's home address. Results:, The geometric mean of total IgE was 161 IU/ml and the average of O3 exposure was 44.9 ± 9.5 ,g/m3. Ozone concentrations were positively related to total IgE levels and an increase of 10 ,g/m3 of O3 resulted in an increase of 20.4% (95% CI = 3.0,40.7) in total IgE levels. Adjustment for age, gender, smoking habits and previous life in the countryside did not change the results, and an increase of 19.1% (2.4,38.6) in total IgE was observed with O3. Negative associations observed between NO2 and total IgE levels disappeared after including O3 in the models. Neither SO2 nor PM10 were correlated with total IgE levels. Conclusions:, Results suggest that O3 or related ambient pollutants may up-regulate total IgE levels among asthmatic adults. [source]

The significance of volcanic eruption strength and frequency for climate

THE QUARTERLY JOURNAL OF THE ROYAL METEOROLOGICAL SOCIETY, Issue 602 2004
G. M. Miles
Abstract A simple physical model of the atmospheric effects of large explosive volcanic eruptions is developed. Using only one input parameter,the initial amount of sulphur dioxide injected into the stratosphere,the global-average stratospheric optical-depth perturbation and surface temperature response are modelled. The simplicity of this model avoids issues of incomplete data (applicable to more comprehensive models), making it a powerful and useful tool for atmospheric diagnostics of this climate forcing mechanism. It may also provide a computationally inexpensive and accurate way of introducing volcanic activity into larger climate models. The modelled surface temperature response for an initial sulphur-dioxide injection, coupled with emission-history statistics, is used to demonstrate that the most climatically significant volcanic eruptions are those of sufficient explosivity to just reach into the stratosphere (and achieve longevity). This study also highlights the fact that this measure of significance is highly sensitive to the representation of the climatic response and the frequency data used, and that we are far from producing a definitive history of explosive volcanism for at least the past 1000 years. Given this high degree of uncertainty, these results suggest that eruptions that release around and above 0.1 Mt SO2 into the stratosphere have the maximum climatic impact. Copyright © 2004 Royal Meteorological Society [source]

Tradable rights to emit air pollution,

AUSTRALIAN JOURNAL OF AGRICULTURAL & RESOURCE ECONOMICS, Issue 1 2009
Dallas Burtraw
The use of cap-and-trade to regulate air pollution promises to achieve environmental goals at lower cost than traditional prescriptive approaches. Cap-and-trade has been applied to various air pollutants including sulphur dioxide, nitrogen oxides and volatile organic compounds in the United States and carbon dioxide (CO2) in the European Union. This corresponds to what is likely to become the most expensive environmental undertaking in history , the effort to reduce the heating of the planet. However, the efficacy of a cap-and-trade policy for CO2 depends in large part on the design of the program. In addition to the level of the cap, the most important decision facing policymakers will be the initial allocation of emissions allowances. The method used to allocate tradable emissions allowances will have significant influence on the distributional impact and efficiency of the program. [source]

Global Energy and Environmental Impacts of an Expanding China

CHINA AND WORLD ECONOMY, Issue 4 2006
Warwick J. McKibbin
Q53; Q56; Q58 Abstract China accounts for 10 percent of global energy use and will continue to rely on coal for generating approximately 75 percent of its energy over coming decades. The environmental problems associated with coal burning are a concern for China as well as regionally and globally. The present paper summarizes China's energy structure and likely future energy requirements, while exploring the impact of energy use on air quality, black carbon emission, sulphur dioxide (SO2) emissions, and carbon dioxide emissions. Although China has begun to take action on local environmental problems from energy, there is still much to be done. In particular, the problem of black carbon and carbon dioxide emissions needs to be addressed. The present paper proposes addressing carbon dioxide emissions through a longer-term strategy that acknowledges the need for China to continue to grow without a short-term carbon constraint but with clear pricing of the short-term and long-term cost of carbon dioxide. (Edited by Xiaoming Feng) [source]

Environmental risk factors and allergic bronchial asthma

CLINICAL & EXPERIMENTAL ALLERGY, Issue 9 2005
G. D'Amato
Summary The prevalence of allergic respiratory diseases such as bronchial asthma has increased in recent years, especially in industrialized countries. A change in the genetic predisposition is an unlikely cause of the increase in allergic diseases because genetic changes in a population require several generations. Consequently, this increase may be explained by changes in environmental factors, including indoor and outdoor air pollution. Over the past two decades, there has been increasing interest in studies of air pollution and its effects on human health. Although the role played by outdoor pollutants in allergic sensitization of the airways has yet to be clarified, a body of evidence suggests that urbanization, with its high levels of vehicle emissions, and a westernized lifestyle are linked to the rising frequency of respiratory allergic diseases observed in most industrialized countries, and there is considerable evidence that asthmatic persons are at increased risk of developing asthma exacerbations with exposure to ozone, nitrogen dioxide, sulphur dioxide and inhalable particulate matter. However, it is not easy to evaluate the impact of air pollution on the timing of asthma exacerbations and on the prevalence of asthma in general. As concentrations of airborne allergens and air pollutants are frequently increased contemporaneously, an enhanced IgE-mediated response to aeroallergens and enhanced airway inflammation could account for the increasing frequency of allergic respiratory allergy and bronchial asthma. Pollinosis is frequently used to study the interrelationship between air pollution and respiratory allergy. Climatic factors (temperature, wind speed, humidity, thunderstorms, etc) can affect both components (biological and chemical) of this interaction. By attaching to the surface of pollen grains and of plant-derived particles of paucimicronic size, pollutants could modify not only the morphology of these antigen-carrying agents but also their allergenic potential. In addition, by inducing airway inflammation, which increases airway permeability, pollutants overcome the mucosal barrier and could be able to ,prime' allergen-induced responses. There are also observations that a thunderstorm occurring during pollen season can induce severe asthma attacks in pollinosis patients. After rupture by thunderstorm, pollen grains may release part of their cytoplasmic content, including inhalable, allergen-carrying paucimicronic particles. [source]