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Sea Otters (sea + otter)
Selected AbstractsCAUSES OF MORTALITY IN CALIFORNIA SEA OTTERS DURING PERIODS OF POPULATION GROWTH AND DECLINEMARINE MAMMAL SCIENCE, Issue 1 2003JamesA. Abstract Elevated mortality appears to be the main reason for both sluggish growth and periods of decline in the threatened California sea otter population. We assessed causes of mortality from salvage records of 3,105 beach-cast carcasses recovered from 1968 through 1999, contrasting two periods of growth with two periods of decline. Overall, an estimated 40%-60% of the deaths were not recovered and 70% of the recovered carcasses died from unknown causes. Nonetheless, several common patterns were evident in the salvage records during the periods of population decline. These included greater percentages of (1) prime age animals (3,10 yr), (2) carcasses killed by great white shark attacks, (3) carcasses recovered in spring and summer, and (4) carcasses for which the cause of death was unknown. Neither sex composition nor the proportion of carcasses dying of infectious disease varied consistently between periods of population increase and decline. The population decline from 1976 to 1984 was likely due to incidental mortality in a set-net fishery, and the decline from 1995 to 1999 may be related to a developing live-fish fishery. Long-term trends unrelated to periods of growth and decline included a decrease in per capita pup production and mass/length ratios of adult carcasses over the 31-yr study. The generally high proportion of deaths from infectious disease suggests that this factor has contributed to the chronically sluggish growth rate of the California sea otter population. [source] LENGTH-MASS AND TOTAL BODY LENGTH OF ADULT FEMALE SEA OTTERS IN PRINCE WILLIAM SOUND BEFORE AND AFTER THE EXXON VALDEZ OIL SPILLMARINE MAMMAL SCIENCE, Issue 4 2002Lisa Mignon Rotterman Abstract After the 1989 T/V Exxon Valdez oil spill (EVOS), the body condition of non-pregnant female sea otters (Enhydra lutris) ages 4 yr and older in the EVOS-affected region of western Prince William Sound, Alaska (WPWS), was significantly poorer than that of individuals captured in the same or adjacent habitat in WPWS approximately a decade earlier, and than that of individuals inhabiting unoiled habitat in eastern PWS (EPWS) between 1984 and 1990. However, the body condition of females of this age category captured in WPWS prior to EVOS was not significantly different from that of pre-and postspill EPWS females. The mean total body length (TBL) of non-pregnant females captured prespill in WPWS was significantly less than that of pre-and postspill EPWS and postspill WPWS females. Evidence from this and other studies suggests that the body condition of at least some classes of sea otters was negatively affected by one or more EVOS-related factors. [source] Loss of genetic diversity in sea otters (Enhydra lutris) associated with the fur trade of the 18th and 19th centuriesMOLECULAR ECOLOGY, Issue 10 2002Shawn Larson Abstract Sea otter (Enhydra lutris) populations experienced widespread reduction and extirpation due to the fur trade of the 18th and 19th centuries. We examined genetic variation within four microsatellite markers and the mitochondrial DNA (mtDNA) d -loop in one prefur trade population and compared it to five modern populations to determine potential losses in genetic variation. While mtDNA sequence variability was low within both modern and extinct populations, analysis of microsatellite allelic data revealed that the prefur trade population had significantly more variation than all the extant sea otter populations. Reduced genetic variation may lead to inbreeding depression and we believe sea otter populations should be closely monitored for potential associated negative effects. [source] LENGTH-MASS AND TOTAL BODY LENGTH OF ADULT FEMALE SEA OTTERS IN PRINCE WILLIAM SOUND BEFORE AND AFTER THE EXXON VALDEZ OIL SPILLMARINE MAMMAL SCIENCE, Issue 4 2002Lisa Mignon Rotterman Abstract After the 1989 T/V Exxon Valdez oil spill (EVOS), the body condition of non-pregnant female sea otters (Enhydra lutris) ages 4 yr and older in the EVOS-affected region of western Prince William Sound, Alaska (WPWS), was significantly poorer than that of individuals captured in the same or adjacent habitat in WPWS approximately a decade earlier, and than that of individuals inhabiting unoiled habitat in eastern PWS (EPWS) between 1984 and 1990. However, the body condition of females of this age category captured in WPWS prior to EVOS was not significantly different from that of pre-and postspill EPWS females. The mean total body length (TBL) of non-pregnant females captured prespill in WPWS was significantly less than that of pre-and postspill EPWS and postspill WPWS females. Evidence from this and other studies suggests that the body condition of at least some classes of sea otters was negatively affected by one or more EVOS-related factors. [source] Loss of genetic diversity in sea otters (Enhydra lutris) associated with the fur trade of the 18th and 19th centuriesMOLECULAR ECOLOGY, Issue 10 2002Shawn Larson Abstract Sea otter (Enhydra lutris) populations experienced widespread reduction and extirpation due to the fur trade of the 18th and 19th centuries. We examined genetic variation within four microsatellite markers and the mitochondrial DNA (mtDNA) d -loop in one prefur trade population and compared it to five modern populations to determine potential losses in genetic variation. While mtDNA sequence variability was low within both modern and extinct populations, analysis of microsatellite allelic data revealed that the prefur trade population had significantly more variation than all the extant sea otter populations. Reduced genetic variation may lead to inbreeding depression and we believe sea otter populations should be closely monitored for potential associated negative effects. [source] The History of Toxoplasma gondii,The First 100 YearsTHE JOURNAL OF EUKARYOTIC MICROBIOLOGY, Issue 6 2008JITENDER P. DUBEY ABSTRACT. In this paper the history of Toxoplasma gondii and toxoplasmosis is reviewed. This protozoan parasite was first discovered in 1908 and named a year later. Its medical importance remained unknown until 1939 when T. gondii was identified in tissues of a congenitally infected infant, and veterinary importance became known when it was found to cause abortion storms in sheep in 1957. The discovery of a T. gondii specific antibody test, Sabin,Feldman dye test in 1948 led to the recognition that T. gondii is a common parasite of warm-blooded hosts with a worldwide distribution. Its life cycle was not discovered until 1970 when it was found that felids are its definitive host and an environmentally resistant stage (oocyst) is excreted in feces of infected cats. The recent discovery of its common infection in certain marine wildlife (sea otters) indicates contamination of our seas with T. gondii oocysts washed from land. Hygeine remains the best preventive measure because currently there is no vaccine to prevent toxoplasmosis in humans. [source] |