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Nasolacrimal Duct (nasolacrimal + duct)
Terms modified by Nasolacrimal Duct Selected AbstractsNasolacrimal Duct Orifice Cysts in Adults: A Previously Unrecognized, Easily Treatable Cause of EpiphoraTHE LARYNGOSCOPE, Issue 10 2007John M. DelGaudio MD Abstract Background: Epiphora is a common problem evaluated by ophthalmologists and otolaryngologists. It is typically the result of obstruction at some level of the nasolacrimal system, either the canaliculi, sac, or duct. Multiple etiologies exist, including scarring from infection or trauma, tumors, or masses. Cysts of the nasolacrimal duct orifice (dacryocystoceles) in the inferior meatus have been described in neonates, usually presenting as obstructive nasal masses shortly after birth. Nasolacrimal duct orifice cysts have not been described in the adult population in the medical literature. Patients: Three patients were identified with epiphora as a result of cysts in the inferior meatus at the opening of the nasolacrimal duct. All patients presented with constant epiphora and were referred for dacryocystorhinostomy by an ophthalmologist or an otolaryngologist. None of the patients had a previous history of nasolacrimal duct (NLD) surgery. One patient had previous endoscopic sinus surgery for nasal polyps. Cysts were identified by nasal endoscopy of the inferior meatus in all patients. Results: All patients underwent endoscopic resection of the inferior meatus cyst to relieve the obstruction of the NLD. Two procedures were performed under general anesthesia and one under intravenous sedation. All patients had complete relief of epiphora and have had no evidence of recurrence of the symptoms or the cyst in 4 to 10 months follow-up. Conclusions: NLD orifice cysts are easily correctable causes of epiphora. Routine inferior meatus endoscopy should be routinely performed in patients with epiphora to identify whether on not this pathology is present prior to performing dacryocystorhinostomy. [source] 3221: Pathophysiology of dry eye syndromeACTA OPHTHALMOLOGICA, Issue 2010J HORWATH-WINTER Dry eye or dysfunctional tear syndrome is a highly prevalent disease worldwide. It is related to a pathological condition of anyone of the parts of the "ocular surface system" that involves the cornea, conjunctiva, lacrimal gland, accessory lacrimal glands, nasolacrimal duct and the lids with the meibomian glands. These are linked as a functional system by innervation, the endocrine and immune system. Endogenous or exogenous caused alterations in one or several components of the ocular surface system or its secretions result in changes of the tear film or ocular surface provoking inflammation. With time, inflammatory reactions may lead to corneal neuropathy compromising the reflex response of the lacrimal glands. Additionally a self-perpetuating vicious circle with loss of function and damage can be initiated also by an immune-modulated inflammation. [source] Correlation between anthropometric facial features and characteristics of nasolacrimal drainage system in connection to false passageCLINICAL & EXPERIMENTAL OPHTHALMOLOGY, Issue 7 2007Junji Narioka MD Abstract Background:, To determine the correlation between the anthropometric facial features and the anatomy of the nasolacrimal drainage system. Methods:, Forty-six mid-sagittal halves of 23 cadavers were studied. The inclination of the lacrimal sac (LS) and nasolacrimal duct (NLD), and the LS-NLD angle were measured in both the sagittal and coronal planes. For the facial features, the height, length and depth of the nose, the intercanthal distance, nasal breadth, nasofacial angle, distance between the external naris, and the inferior aperture of the NLD were measured. The correlations between the angles and the facial features were calculated. Results:, The LS-NLD angle was classified into three anatomical types according to the inclination of the LS and the NLD in the sagittal and coronal planes; the anterior-inward type (43.5%), anterior-outward type (37.0%), and posterior-outward type (19.5%). The nasal depth was correlated with the sagittal LS-NLD angle, and the nasal height and length were correlated with the coronal LS-NLD angle. Conclusions:, There is a large variation in the LS-NLD angle, and nasal depth, height and length can be used to predict the direction of this angle. [source] The lacrimal fascia redefinedCLINICAL ANATOMY, Issue 6 2001Harumichi Shinohara Abstract It has been commonly accepted that the lacrimal fascia is an intact septum, composed of connective tissue, that separates the orbital cavity into a large compartment, which contains the eyeball, and a small compartment, which contains the lacrimal sac and nasolacrimal duct. the septum, however, is not necessarily always intact. We found a circular or oval opening in the lacrimal fascia in 37 of 52 specimens (71.2%) examined. The opening, which we shall refer to as the lacrimal fascial foramen, was located at variable levels in the lacrimal fossa. The lacrimal fascial foramen was buried in fatty tissue through which passed a branch of either the inferior palpebral artery or the infraorbital artery and a vein of the nasolacrimal duct. The clinical implications of the lacrimal fascial foramen in obstruction of the nasolacrimal duct are discussed. Clin. Anat. 6:401,405, 2001. © 2001 Wiley-Liss, Inc. [source] Erosive mucosal lichen planus and secondary epiphora responding to systemic cyclosporin A treatmentAUSTRALASIAN JOURNAL OF DERMATOLOGY, Issue 3 2009Aaron E Boyce ABSTRACT Erosive mucosal lichen planus (LP) is a well-established variant of LP characterized by the formation of ulcerative lesions predominantly involving the oral and genital mucosae. Less commonly, this condition may involve oesophageal and/or ocular mucosal surfaces, and case reports within the ophthalmology literature have recently confirmed the potential for this condition to affect the nasolacrimal ducts. We report the case of a woman with severe cicatrizing mucosal LP and ocular symptoms secondary to presumed nasolacrimal duct involvement. We also report the potential for this newly appreciated manifestation of LP to respond to systemic cyclosporin A. [source] |