Green Peach Aphid (green + peach_aphid)

Distribution by Scientific Domains

Terms modified by Green Peach Aphid

  • green peach aphid Myzu persicae

  • Selected Abstracts


    STUDIES ON SELECTIVE TOXICITY OF SIX INSECTICIDES BETWEEN GREEN PEACH APHID AND LADYBIRDS

    INSECT SCIENCE, Issue 2 2002
    XUE Ming
    Abstract, The selective toxicity of six kinds of insecticides, including imidacloprid, imidacloprid + synergist (SV1), fenvalerate, endosulfan, methomyl and dimethoate, between the green peach aphid (Myzus persicae Sulzer) and two species of ladybirds (Coccinella septempunctata Linnaeus and Propylaea japonica Thunbery), was investigated in the laboratory. The reults showed that both imidacloprid WP and imidacloprid + synergist (SVl) EC possessed the highest toxicity to the aphids. Between C. septempunctata and M. persicae and between P. japonica and M. persicae, the selective toxicity ratios (STRs) of imidacloprid WP, imidacloprid+ synergist (SV1) EC and endosulfan EC were 37.6 and 13.0, 9.84 and 7.75, 54.0 and 7.28 respectively. All of them showed rather high selective toxicity. The STRs of fenvalerate EC, dimethoate EC and methomyl EC were all very low, ranging from 0.02 to 0.21, indicating their low degree of safety to the two species of ladybids. The results demomarated that imidacloprid WP and imidacloprid + SVl EC not only had rather high toxicity to the aphids, but also reduced strikingly the reproduction rate and fecundity of the survival aphids. Insecticides can induce the relative fitness of insects decrease. Among the six insecticides tested with M. persicae, the following were insecticides and the order of induction was: imidacloprid + SV1 imidacloprid endosulfan methomyl fenvalerate > dimethoate. [source]


    Green peach aphid, Myzus persicae (Hemiptera: Aphididae), reproduction during exposure to sublethal concentrations of imidacloprid and azadirachtin

    PEST MANAGEMENT SCIENCE (FORMERLY: PESTICIDE SCIENCE), Issue 2 2009
    G Christopher Cutler
    Abstract BACKGROUND: Resurgence of insect pests following insecticide applications is often attributed to natural enemy disturbance, but hormesis could be an alternative or additional mechanism. Green peach aphid, Myzus persicae (Sulzer), is an important insect pest of many crops worldwide that may be exposed to sublethal insecticide concentrations over time. Here, the hypothesis that exposure to low concentrations of imidacloprid and azadirachtin can induce hormetic responses in M. persicae is tested in the laboratory. RESULTS: When insects were exposed to potato leaf discs dipped in sublethal concentrations of insecticide, almost all measured endpoints,adult longevity, F1 production, F1 survival and F2 production,were affected, and a statistically significant (P < 0.05) stimulatory response was recorded for F2 production following exposure to imidacloprid. No other measures for hormesis were statistically significant, but other trends of hormetic response were consistently observed. CONCLUSIONS: Given that variable distribution and degradation of insecticides in the field would result in a wide range of concentrations over time and space, these laboratory experiments suggest that exposure to sublethal concentrations of imidacloprid and azadirachtin could stimulate reproduction in M. persicae. Copyright © 2008 Society of Chemical Industry [source]


    Impact of chemical elicitor applications on greenhouse tomato plants and population growth of the green peach aphid, Myzus persicae

    ENTOMOLOGIA EXPERIMENTALIS ET APPLICATA, Issue 3 2006
    Anthony J. Boughton
    Abstract Recent advances in the understanding of plant signaling pathways have opened the way for using elicitor-induced plant resistance as a tactic for protecting plants against arthropod pests. Four common elicitors of induced responses in tomato, Lycopersicon esculentum Mill. (Solanaceae), were evaluated with regard to phytotoxicity, induction of plant defensive proteins, and effects on population growth and fecundity of a common pest, the green peach aphid, Myzus persicae (Sulzer) (Homoptera: Aphididae). Ethephon and methyl jasmonate (MJ) treatments caused varying degrees of phytotoxicity. Ethephon caused pronounced changes in plant growth form and severe, dose-dependent negative impacts on plant growth and flowering. Effects with MJ were milder, but still caused temporary inhibition of development, leading to smaller plants and delayed flowering. The commercial elicitors benzothiadiazole (BTH) and harpin did not cause detectable phytotoxicity. The highest doses of ethephon and MJ significantly increased leaf peroxidase (POD) levels but only MJ treatments significantly increased polyphenol oxidase (PPO) levels. BTH and harpin had no detectable effects on POD and PPO. Populations of green peach aphids grew significantly more slowly on plants treated with BTH or MJ than on control plants or plants treated with harpin or ethephon. Slowed aphid population growth on BTH-treated plants was due to significant reductions in aphid fecundity, although this was independent of changes in time to onset of reproduction or time to death. Aphid fecundity was also reduced on MJ-treated plants relative to controls, but this difference was not statistically significant, suggesting that other mechanisms are involved in slowing aphid population growth on MJ-treated plants. Growth of aphid populations on plants treated with a MJ,BTH mixture was reduced almost as much as with treatments of MJ alone, suggesting that antagonism between JA-dependant and SA-dependent plant signaling pathways is only mild with regard to induced defenses against aphids. [source]


    Studies on the morphometry of the green peach aphid, Myzus persicae (Sulzer) (Homoptera: Aphididae)

    ENTOMOLOGICAL RESEARCH, Issue 2 2007
    Potshangbam Bijaya DEVI
    Abstract Aphids are one of the most important groups of phytophagous insects because of their polyphenism, host alternation, heteroecious behavior and reproductive habits. Laboratory culture of the aphid Myzus persicae on the host plant Brassica pekinensis Hubner was maintained seasonally for biometrical studies. The metric analysis revealed gradual developmental changes from the first instar nymph to the adult stage in size, shape, and a number of characters. The transitional changes were sufficiently distinct to allow differentiation of each of the life stages irrespective of the season. A key to the nymphal stages of the pest is provided. [source]


    STUDIES ON SELECTIVE TOXICITY OF SIX INSECTICIDES BETWEEN GREEN PEACH APHID AND LADYBIRDS

    INSECT SCIENCE, Issue 2 2002
    XUE Ming
    Abstract, The selective toxicity of six kinds of insecticides, including imidacloprid, imidacloprid + synergist (SV1), fenvalerate, endosulfan, methomyl and dimethoate, between the green peach aphid (Myzus persicae Sulzer) and two species of ladybirds (Coccinella septempunctata Linnaeus and Propylaea japonica Thunbery), was investigated in the laboratory. The reults showed that both imidacloprid WP and imidacloprid + synergist (SVl) EC possessed the highest toxicity to the aphids. Between C. septempunctata and M. persicae and between P. japonica and M. persicae, the selective toxicity ratios (STRs) of imidacloprid WP, imidacloprid+ synergist (SV1) EC and endosulfan EC were 37.6 and 13.0, 9.84 and 7.75, 54.0 and 7.28 respectively. All of them showed rather high selective toxicity. The STRs of fenvalerate EC, dimethoate EC and methomyl EC were all very low, ranging from 0.02 to 0.21, indicating their low degree of safety to the two species of ladybids. The results demomarated that imidacloprid WP and imidacloprid + SVl EC not only had rather high toxicity to the aphids, but also reduced strikingly the reproduction rate and fecundity of the survival aphids. Insecticides can induce the relative fitness of insects decrease. Among the six insecticides tested with M. persicae, the following were insecticides and the order of induction was: imidacloprid + SV1 imidacloprid endosulfan methomyl fenvalerate > dimethoate. [source]


    Sublethal effects of selected insecticides on fecundity and wing dimorphism of green peach aphid (Hom., Aphididae)

    JOURNAL OF APPLIED ENTOMOLOGY, Issue 2 2008
    X.-Y. Wang
    Abstract Effects of sublethal concentrations (LC25) of six insecticides (imidacloprid, rotenone, fenvalerate, abamectin, pirimicarb and azadirachtin) on fecundity and wing dimorphism of the green peach aphid, Myzus persicae (Sulzer), were studied both under laboratory and greenhouse conditions. In the laboratory, aphid reproduction reduced by 44.29% and 54.01% when rotenone and abamectin treatments were applied at sublethal dose, respectively, and sublethal fenvalerate application resulted in markedly lower average reproduction per female per day compared with control. Reproductive duration of aphid treated with abamectin significantly decreased by 44.19%. But in the greenhouse, there were no evident differences in the aphid fecundity and reproductive duration between treatments and control. Life-table parameters also demonstrated that the six insecticides at sublethal doses did not stimulate the aphid reproductive potential. In the laboratory, after being exposed to sublethal doses of imidacloprid and fenvalerate, the proportions of alate progeny in aphid progeny were significantly higher than that of the control. In the greenhouse, percentages of alate offspring from the mother aphids treated with imidacloprid, fenvalerate and abamectin increased pronouncedly compared with control. Mortality rates of offspring in the nymphal stages from adults treated with insecticides revealed no significant changes between laboratory and greenhouse. The developmental time in days of the offspring varied in all treatments. Mechanisms of insecticide-induced resurgence are discussed. [source]


    Monitoring for imidacloprid resistance in the tobacco-adapted form of the green peach aphid, Myzus persicae (Sulzer) (Hemiptera: Aphididae), in the eastern United States

    PEST MANAGEMENT SCIENCE (FORMERLY: PESTICIDE SCIENCE), Issue 6 2010
    Lakshmipathi Srigiriraju
    Abstract BACKGROUND: Imidacloprid is the primary insecticide for controlling the tobacco-adapted form of the green peach aphid (TGPA), Myzus persicae (Sulzer), a major pest of tobacco worldwide. This study used leaf-dip bioassays to assess TGPA resistance to imidacloprid in the eastern United States from 2004 through 2007. RESULTS: When combined over the 4 year study, 18, 14 and 3% of the TGPA had imidacloprid resistance ratios (RRs) of 10,20-fold, 20,30-fold and 30,90-fold, respectively, compared with the most susceptible colony tested. This indicates that some colonies have developed moderate levels of resistance to imidacloprid. A colony collected near Clayton, North Carolina, had the highest RR of 91 (LC50 value = 31 mg L,1). This resistance declined for six tests over a 3 year period in the laboratory culture from >130-fold RR (LC50 = 48 mg L,1) to 40-fold RR (LC50 = 15 mg L,1). Over the same period, the most susceptible colony and a standard colony not exposed to imidacloprid for over 7 years had consistently low LC50 values. CONCLUSION: Moderate levels of resistance to imidacloprid are noticed among TGPA colonies from the eastern United States. The variation in resistance indicates that the factors responsible are present in the populations at low frequencies and are just not enough to cause field failures yet. Copyright © 2010 Society of Chemical Industry [source]


    Esterase-based resistance in the tobacco-adapted form of the green peach aphid, Myzus persicae (Sulzer) (Hemiptera: Aphididae) in the eastern United States

    ARCHIVES OF INSECT BIOCHEMISTRY AND PHYSIOLOGY (ELECTRONIC), Issue 2 2009
    Lakshmipathi Srigiriraju
    Abstract Organophosphates and carbamates represent alternative insecticides in managing the tobacco-adapted form of the green peach aphid (TGPA), Myzus persicae (Sulzer), a major pest of tobacco in the United States and around the world. General esterases that detoxify these insecticides were assessed in green, red, and orange morphs of field-collected M. persicae. A total of 136 aphid colonies were collected from 2004 though 2007 and screened for total esterase activity. The green morphs had lower esterase levels, with a mean of 77±6.6,nmol/min/mg protein, as compared to red (84±2.9,nmol/min/mg protein) and orange morphs (172±16.5,nmol/min/mg protein). Overall esterase activities, and those for the red and green morphs, were positively correlated with LC50 values for acephate (organophosphate) and methomyl (carbamate) assessed in leaf-dip bioassays. Esterase genes responsible for higher esterase activities were diagnosed by gene amplification studies. Twenty-three of 24 colonies tested had either the E4 or FE4 gene amplified, both known to confer esterase-based resistance. Fifteen out of the 24 colonies tested had amplified E4 gene and four colonies had FE4 gene amplification. All orange morphs and one green morph had both E4 and FE4 genes amplified. This unique phenotype, where two esterase genes were amplified had an 865-bp band characteristic of the FE4 gene and an additional 381-bp band characteristic of a deleted upstream region of the E4 gene. Changes that occurred in esterase-based resistance in the TGPA over the past two decades and their implications on insecticide resistance management are discussed. © 2009 Wiley Periodicals, Inc. [source]


    Impact of chemical elicitor applications on greenhouse tomato plants and population growth of the green peach aphid, Myzus persicae

    ENTOMOLOGIA EXPERIMENTALIS ET APPLICATA, Issue 3 2006
    Anthony J. Boughton
    Abstract Recent advances in the understanding of plant signaling pathways have opened the way for using elicitor-induced plant resistance as a tactic for protecting plants against arthropod pests. Four common elicitors of induced responses in tomato, Lycopersicon esculentum Mill. (Solanaceae), were evaluated with regard to phytotoxicity, induction of plant defensive proteins, and effects on population growth and fecundity of a common pest, the green peach aphid, Myzus persicae (Sulzer) (Homoptera: Aphididae). Ethephon and methyl jasmonate (MJ) treatments caused varying degrees of phytotoxicity. Ethephon caused pronounced changes in plant growth form and severe, dose-dependent negative impacts on plant growth and flowering. Effects with MJ were milder, but still caused temporary inhibition of development, leading to smaller plants and delayed flowering. The commercial elicitors benzothiadiazole (BTH) and harpin did not cause detectable phytotoxicity. The highest doses of ethephon and MJ significantly increased leaf peroxidase (POD) levels but only MJ treatments significantly increased polyphenol oxidase (PPO) levels. BTH and harpin had no detectable effects on POD and PPO. Populations of green peach aphids grew significantly more slowly on plants treated with BTH or MJ than on control plants or plants treated with harpin or ethephon. Slowed aphid population growth on BTH-treated plants was due to significant reductions in aphid fecundity, although this was independent of changes in time to onset of reproduction or time to death. Aphid fecundity was also reduced on MJ-treated plants relative to controls, but this difference was not statistically significant, suggesting that other mechanisms are involved in slowing aphid population growth on MJ-treated plants. Growth of aphid populations on plants treated with a MJ,BTH mixture was reduced almost as much as with treatments of MJ alone, suggesting that antagonism between JA-dependant and SA-dependent plant signaling pathways is only mild with regard to induced defenses against aphids. [source]