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Venous Outflow (venous + outflow)
Terms modified by Venous Outflow Selected AbstractsVascular pedicle avulsion in free flap breast reconstruction: A case of diep flap salvage following early avulsion of venous anastomosis and literature reviewMICROSURGERY, Issue 3 2010Efstathios G. Lykoudis M.D., Ph.D. Free flap vascular pedicle avulsion represents an extremely rare complication in reconstructive microsurgery. Very few cases have been reported in the literature, most of them identified in free flap breast reconstruction. As a result, little data is currently available on the etiology and treatment of this rare complication. Herein, we report a unique case of early venous anastomosis avulsion following free DIEP flap transfer for delayed breast reconstruction. Venous outflow was successfully restored with the use of an interposition vein graft, and the flap survived completely. In addition, the relevant literature is reviewed; and the possible causes, preventive strategies, and management options are analyzed. © 2010 Wiley-Liss, Inc. Microsurgery 2010. [source] Mechanical prophylaxis for travellers' thrombosis: a comparison of three interventions that promote venous outflowJOURNAL OF THROMBOSIS AND HAEMOSTASIS, Issue 7 2007M. COPPENS No abstract is available for this article. [source] The Effect of Rigid Cervical Collars on Internal Jugular Vein DimensionsACADEMIC EMERGENCY MEDICINE, Issue 1 2010Michael B. Stone MD Abstract Objectives:, Prior research has demonstrated that rigid cervical collars cause an increase in intracranial pressure (ICP). The mechanism for this effect is unclear and one proposed mechanism involves obstruction of venous outflow in the neck. Ultrasound (US) allows assessment of internal jugular vein dimensions and may yield information regarding the mechanism for the increase in ICP seen with rigid collar application. Methods:, Forty-two healthy volunteers underwent US examination of the internal jugular vein before and after cervical collar application. Internal jugular vein cross-sectional areas were compared with and without the cervical collar in place. Results:, The cross-sectional area of the internal jugular vein increased significantly (p < 0.0001) after application of the cervical collar. The mean percentage increase in cross-sectional area was 37% (95% confidence interval [CI] = 20% to 53%). Conclusions:, Internal jugular vein cross-sectional area increases after application of a rigid cervical collar. This supports the hypothesis that venous obstruction in the neck may contribute to the increase in ICP seen after rigid collar application. ACADEMIC EMERGENCY MEDICINE 2010; 17:100,102 © 2009 by the Society for Academic Emergency Medicine [source] Management of flaps with compromised venous outflow in head and neck microsurgical reconstructionMICROSURGERY, Issue 8 2002Tateki Kubo M.D. Microvascular tissue transfer has become an indispensable procedure for head and neck reconstruction. Although remarkable progress has been made technically, anastomosed vessel occlusion is still a serious complication. Even with technically skilled microsurgeons, anastomosed vessel occlusion occurs because the technique is not the sole prophylaxis against thrombosis in microsurgery. Therefore, to minimize the possibility of an unfavorable result in microsurgery, microsurgeons must be familiar with management options for a vascular compromised flap. Most investigators have agreed that venous obstruction occurs more often than arterial obstruction. Here, we reviewed the published literature on the salvage of venous compromised flaps from the viewpoints of surgical correction, including reanastomosis and catheter thrombectomy, and nonsurgical procedures, such as a medicinal leech, hyperbaric oxygen, and thrombolytic therapy. © 2002 Wiley-Liss, Inc. MICROSURGERY 22:391,395 2002 [source] Extracting arterial flow waveforms from pulse oximeter waveformsANAESTHESIA, Issue 6 2001apparatus A method is described which allows an approximation to the arterial flow waveform to be derived from a pulse oximeter waveform. The observed pulse oximeter waveform is the sum of arterial inflow and venous outflow. These components are separated mathematically. Subtraction of the venous outflow reveals the underlying arterial flow waveform. The assumptions on which the method is based are stated explicitly and discussed. [source] Venous and cerebrospinal fluid flow in multiple sclerosis: A case-control studyANNALS OF NEUROLOGY, Issue 2 2010Peter Sundström MD The prevailing view on multiple sclerosis etiopathogenesis has been challenged by the suggested new entity chronic cerebrospinal venous insufficiency. To test this hypothesis, we studied 21 relapsing-remitting multiple sclerosis cases and 20 healthy controls with phase-contrast magnetic resonance imaging. In addition, in multiple sclerosis cases we performed contrast-enhanced magnetic resonance angiography. We found no differences regarding internal jugular venous outflow, aqueductal cerebrospinal fluid flow, or the presence of internal jugular blood reflux. Three of 21 cases had internal jugular vein stenoses. In conclusion, we found no evidence confirming the suggested vascular multiple sclerosis hypothesis. ANN NEUROL 2010;68:255,259 [source] Late Symptomatic Venous Stenosis in Three Hemodialysis Patients Without Previous Central Venous CathetersARTIFICIAL ORGANS, Issue 12 2000Massimo Morosetti Abstract: It is well known that catheters placed in the subclavian or internal jugular veins may develop stenosis in the vein in which the catheter lies. Because the arteriovenous fistula (AVF) relies on good venous outflow, patients with ipsilateral central venous stenosis are subject to the malfunctioning of AVF. Until now, no data were published on patients showing central vein stenosis (CVS) without a previous central venous catheter (CVC) or a pacemaker. In this article, we report on 3 hemodialysis patients manifesting CVS ipslateral to AVF. None of these patients previously had undergone CVC. The stenosis observed had characteristics and symptoms similar to those observed in stenoses consequent to CVC. We concluded that CVS also may occur in subclavian or axillary veins proximal to a working AVF in hemodialysis patients who have never had a CVC and in the absence of compressive phenomena. [source] Hepatic venous outflow reconstruction in adult living donor liver transplants without portal hypertensionCLINICAL TRANSPLANTATION, Issue 2 2004Diego Bogetti Abstract:, Graft congestion is one of the causes of poor graft function in segmental liver transplantation. Three factors are implicated in segmental graft congestion: graft size, hepatic venous outflow and portal inflow. The graft size must be matched to the body weight, which is conventionally done by using graft to body weight ratio. Hepatic blood outflow must be optimized by hepatic vein reconstruction, which can be complicated. High portal blood flow has been shown to be detrimental to small-for-size grafts. These factors are strictly connected to each other. They can all contribute to graft congestion and poor function, while one factor can compensate for the others and decrease congestion. Ideally, all the accessory veins should be reconstructed, if possible, to maximize the outflow. In the absence of portal hypertension and with an adequate sized graft, complex venous reconstruction may not be necessary. We present a case report of an adult living donor liver transplant with the favorable conditions of normal portal pressure and a large sized graft, but complicated by the presence of several accessory hepatic veins. A simple hepatic vein anastomosis was sufficient for adequate outflow and prompt graft function. [source] |