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Movement Disorders (movement + disorders)

Distribution by Scientific Domains
Medical Sciences92%
Life Sciences4%
2 Other Domains4%
Distribution within Medical Sciences
Neurology91%
Psychiatry2%
6 Other Subdomains7%

Kinds of Movement Disorders

  • other movement disorders
    		•
  • psychogenic movement disorders
    		•
  • various movement disorders
    		•

    Terms modified by Movement Disorders

  • movement disorders clinic
    		•

    Selected Abstracts

    Psychogenic Movement Disorders: Neurology and Psychiatry

    EUROPEAN JOURNAL OF NEUROLOGY, Issue 11 2006
    K. A. Jellinger
    No abstract is available for this article. [source]

    Drug Induced Movement Disorders, 2nd edition

    EUROPEAN JOURNAL OF NEUROLOGY, Issue 9 2006
    K. A. Jellinger
    No abstract is available for this article. [source]

    Mental and Behavioral Dysfunction in Movement Disorders

    EUROPEAN JOURNAL OF NEUROLOGY, Issue 4 2003
    K. A. Jellinger
    No abstract is available for this article. [source]

    Modeling Effective Treatment of Psychoses in the Elderly: A Focus on Avoiding Movement Disorders

    JOURNAL OF AMERICAN GERIATRICS SOCIETY, Issue 2004
    Richard M. Trosch MD
    No abstract is available for this article. [source]

    Abstracts of the Fourteenth International Congress of Parkinson's Disease and Movement Disorders

    MOVEMENT DISORDERS, Issue S2 2010
    Article first published online: 20 MAY 2010
    First page of article [source]

    Program: Twenty Fourth Annual Symposium on Etiology, Pathogenesis, and Treatment of Parkinson's Disease and Other Movement Disorders

    MOVEMENT DISORDERS, Issue 6 2010
    Article first published online: 26 APR 2010
    The symposium will consist of current issues in genetic and environmental contributions to Parkinson's disease and other movement disorders with peer-reviewed platform and poster presentations designed to communicate recent research advances, including new pharmacological and non-pharmacological treatment options, in the field of Parkinson's disease, Huntington's disease, ataxia, dystonia, myoclonus, Tourette's syndrome, tremor and other movement disorders thereby enhancing patient care. [source]

    Program: Twenty Third Annual Symposium on Etiology, Pathogenesis, and Treatment of Parkinson's Disease and Other Movement Disorders

    MOVEMENT DISORDERS, Issue 12 2009
    Article first published online: 11 SEP 200
    The symposium will consist of two keynote speakers and peer-reviewed platform and poster presentations designed to communicate recent research advances, including new pharmacological and non-pharmacological treatment options, in the field of Parkinson's disease, Huntington disease, ataxia, dystonia, myoclonus, Tourette's syndrome, Essential Tremor and other movement disorders thereby enhancing patient care. Professionals in neurology and related disciplines as well as practitioners, psychologists, educators, and researchers are invited to attend. The gaps in clinical practice we wish to address are the unmet needs pertaining to the translational and clinical evaluation, along with the care and treatment of patients and families affected by Parkinson's disease and other movement disorders. At the conclusion of this session, participants should be able to: 1) Identify and describe by scholarly review, oral presentation and group discussion the current research into the diagnosis, prevention and treatment of Parkinson's disease (PD) and Essential Tremor (ET) which may be relevant to current treatment or which may lead to the development of further research protocols; 2) Distinguish and assess the important advances in research and clinical treatments relating to Parkinson's disease and Essential Tremor in terms of available treatment options or new methodologies for clinical research; 3) Explain new pharmacological and non-pharmacological treatment options available for Parkinson's disease and other movement disorders in connection with their clinical practice or with regard to further clinical research methods; 4) Interpret the mechanisms (genetic, environmental, pathophysiology, neurobiology) linked to Parkinson's disease and other movement disorders when assessing Parkinson's disease or other movement disorder patients or when developing new research protocols; and 5) Employ diagnostic approaches and tools available for assessing Parkinson's disease and Essential Tremor when diagnosing new patients or when conducting clinical research. [source]

    Abstracts of The Movement Disorder Society's Thirteenth International Congress of Parkinson's Disease and Movement Disorders

    MOVEMENT DISORDERS, Issue S1 2009
    Article first published online: 27 MAY 200
    [source]

    Abstracts of The Movement Disorder Society's Twelfth International Congress of Parkinson's Disease and Movement Disorders

    MOVEMENT DISORDERS, Issue S1 2008
    Article first published online: 28 MAY 200
    [source]

    Abstracts of The Movement Disorder Society's Eleventh International Congress of Parkinson's Disease and Movement Disorders

    MOVEMENT DISORDERS, Issue S16 2007
    Article first published online: 24 APR 200
    [source]

    Current controversies: Levodopa in the treatment of Parkinson's disease

    MOVEMENT DISORDERS, Issue 5 2005
    Jagdish C. Sharma FRCP
    Companion letters have been published in Movement Disorders: Levodopa in the Treatment of Parkinson's Disease: Current Controversies, by Gerlach, Reichmann, and Riederer and Reply: Levodopa in the Treatment of Parkinson's Disease, by Olanow, Agid, and Mizuno. [source]

    Movement Disorders, Volume 20, Supplement 10, 2005: Index of Authors

    MOVEMENT DISORDERS, Issue S10 2005
    Article first published online: 9 FEB 200
    [source]

    Valvular heart disease and fibrotic reactions may be related to ergot dopamine agonists, but non-ergot agonists may also not be spared

    MOVEMENT DISORDERS, Issue 12 2004
    FRCP, K. Ray Chaudhuri MD
    Companion letters have been published in this issue of Movement Disorders: Horowski et al., pp 1523,1524, and Rascol et al., pp 1524,1525. [source]

    Fibrotic valvular heart disease is not related to chemical class but to biological function: 5-HT2B receptor activation plays crucial role

    MOVEMENT DISORDERS, Issue 12 2004
    Reinhard Horowski MD
    Companion letters have been published in this issue of Movement Disorders: Chaudhuri et al., pp 1522,1523, and Rascol et al., pp 1524,1525. [source]

    Transplantation in Parkinson's disease: PET changes correlate with the amount of grafted tissue

    MOVEMENT DISORDERS, Issue 8 2003
    Valérie Cochen MD
    Abstract An erratum for this article appears in the January, 2004 issue of Movement Disorders (Mov Disord 2004;12:119). We compared the striatal uptake of [18F]fluorodopa with [76Br]-FE-CBT, a positron emission tomography (PET) ligand of the dopamine transporter (DAT), which estimates the density of dopamine nerve terminals, in 6 patients with Parkinson's disease grafted with fetal mesencephalic cells. There was no change in DAT ligand binding in the grafted putamen, despite a significant increase of [18F]fluorodopa uptake. This finding suggests that the clinical benefit induced by the graft is more related to increased dopaminergic activity than improved dopaminergic innervation in the host striatum and, therefore, that [18F]fluorodopa remains the optimal tracer to evaluate grafted PD patients. Further analysis showed that the clinical and [18F]fluorodopa uptake changes after the grafts were correlated with the number of ventral mesencephalae used for implantation. © 2003 Movement Disorder Society [source]

    Pathogenic role of glial cells in Parkinson's disease

    MOVEMENT DISORDERS, Issue 2 2003
    Peter Teismann PhD
    Abstract An erratum for this article appears in the January, 2004 issue of Movement Disorders (Mov Disord 2004;19:119). Parkinson's disease (PD) is a common neurodegenerative disorder characterized by the progressive loss of the dopaminergic neurons in the substantia nigra pars compacta (SNpc). The loss of these neurons is associated with a glial response composed mainly of activated microglial cells and, to a lesser extent, of reactive astrocytes. This glial response may be the source of trophic factors and can protect against reactive oxygen species and glutamate. Alternatively, this glial response can also mediate a variety of deleterious events related to the production of pro-oxidant reactive species, and pro-inflammatory prostaglandin and cytokines. We discuss the potential protective and deleterious effects of glial cells in the SNpc of PD and examine how those factors may contribute to the pathogenesis of this disease. © 2002 Movement Disorder Society [source]

    American Neurological Association, 128th Annual Meeting Abstracts: Poster Sessions (Movement Disorders)

    ANNALS OF NEUROLOGY, Issue S7 2003
    Article first published online: 28 AUG 200
    First page of article [source]

    Amphetamine-induced movement disorder

    EMERGENCY MEDICINE AUSTRALASIA, Issue 3 2005
    Michael A Downes
    Abstract Recreational use of amphetamines is common in Australia and New Zealand when compared with other developed nations. The clinical effects are variable because of the potential of these drugs to increase the proportion of different biogenic amines in the central nervous system (CNS). The substances affected are adrenaline, noradrenaline, serotonin and dopamine. Movement disorders represent one of the less common presentations of amphetamine toxicity but one that health care workers should be aware of nonetheless. [source]

    Transient Athetosis Induced by Tiagabine

    EPILEPSIA, Issue 4 2006
    Mario Tombini
    Summary:, Movement disorders have been reported with use of different antiepileptic drugs (AEDs). We report a 32-year-old woman, affected by a symptomatic focal drug-resistant epilepsy and a mild hemiparesis, with acute athetoid movements, transiently linked to increasing tiagabine (TGB) therapy. To our knowledge, no other cases of acute athetosis related to TGB have been reported to date. However, we cannot rule out the possibility that involuntary movements were induced by an interaction between TGB and concomitant AEDs, in particular phenobarbital (PB), possibly by increasing GABAergic transmission. We hypothesize that the presence of a static encephalopathy may have influenced the kind of extrapyramidal side effect induced by TGB in our patient, leading to athetosis. [source]

    Movement disorders and pregnancy: A review of the literature,,

    MOVEMENT DISORDERS, Issue 6 2010
    Sarah M. Kranick MD
    Abstract Pregnant patients are rarely encountered in the movement disorders clinic, but they present significant dilemmas regarding treatment and counseling for neurologists. While movement disorders in pregnancy once described those disorders arising de novo during pregnancy, such as chorea gravidarum or restless leg syndrome, advancing maternal age in Western countries will likely increase the number of women in whom pregnancy complicates a pre-existing movement disorder. Physicians treating these women must be aware of the impact of the movement disorder and its treatment on fertility, pregnancy, fetal development, lactation, and infant care. This review summarizes retrospective series and case reports to both guide clinicians and to stimulate and direct the design of prospective studies. © 2010 Movement Disorder Society [source]

    Movement disorders in musicians,,

    MOVEMENT DISORDERS, Issue 14 2008
    Joseph Jankovic MD
    Abstract The focus of this article is to review the epidemiology, phenomenology, pathophysiology, genetics, and treatment of movement disorders, particularly task-specific dystonia, in musicians. The goal is to draw attention to this group of neurological disorders among musicians, music teachers, and healthcare professionals and to highlight the importance of early diagnosis, therapeutic options, and preventive measures. To increase professional and public awareness and to facilitate the recognition of music-related neurological problems, we suggest that "medical problems of musicians" be included in curriculum of music schools and medical schools. © 2008 Movement Disorder Society [source]

    Movement disorders associated with hyperthyroidism: Expanding the phenotype

    MOVEMENT DISORDERS, Issue 7 2006
    Eng-King Tan
    [source]

    Movement disorders associated with encephalitis lethargica: A video compilation

    MOVEMENT DISORDERS, Issue 1 2006
    Joel A. Vilensky PhD
    Abstract Encephalitis lethargica (EL; epidemic encephalitis; von Economo's disease) often presented with a movement disorder, and the motor consequences of postencephalitic parkinsonism (PEP) were characteristic of the chronic sequelae of this condition. PEP was similar to Parkinson's disease but was more variable and had some distinct features such as oculogyric crises. Although two previous publications have included video images of the movement disorders associated with EL and PEP, the sequences presented were typically short, showed only a few patients, and did not include the work of several neurologists who had the foresight to preserve filmed images of their patients. We describe the most complete record of EL and PEP moving images that have been preserved and make them available in edited form. © 2005 Movement Disorder Society [source]

    Coexistence of movement disorders and epilepsia partialis continua as the initial signs in probable Creutzfeldt,Jakob disease

    MOVEMENT DISORDERS, Issue 9 2005
    Berril Donmez MD
    Abstract Movement disorders and epilepsy rarely occur in the early stage of Creutzfeldt,Jakob disease (CJD) but have not been reported concurrently. We report on a 47-year-old patient with probable CJD who presented with generalized chorea and focal dystonia with myoclonic jerks on the right hand. Myoclonic jerks progressed to epilepsia partialis continua within 5 days of admission to the hospital. The diagnosis of our patient was compatible with probable CJD on the basis of clinical course, electroencephalogram, and diffusion-weighted magnetic resonance imaging findings, and presence of 14-3-3 protein in cerebrospinal fluid. To our knowledge, this is the first report of a case developing both movement disorders and epilepsia partialis continua in the early stage of the disease. © 2005 Movement Disorder Society [source]

    Movement disorders in patients with peripheral facial palsy,

    MOVEMENT DISORDERS, Issue 12 2003
    Josep Valls-Solé MD
    Abstract Acute unilateral facial paralysis is usually a benign neurological condition that resolves in a few weeks. However, it can also be the source of a transient or long-lasting severe motor dysfunction, featuring disorders of automatic and voluntary movement. This review is organized according to the two most easily recognizable phases in the evolution of facial paralysis: (1) Just after presentation of facial palsy, patients may exhibit an increase in their spontaneous blinking rate as well as a sustained low-level contraction of the muscles of the nonparalyzed side, occasionally leading to blepharospasm-like muscle activity. This finding may be due to an increase in the excitability of facial motoneurons and brainstem interneurons mediating trigeminofacial reflexes. (2) If axonal damage has occurred, axonal regeneration beginning at approximately 3 months after the lesion leads inevitably to clinically evident or subclinical hyperactivity of the previously paralyzed hemifacial muscles. The full-blown postparalytic facial syndrome consists of synkinesis, myokymia, and unwanted hemifacial mass contractions accompanying normal facial movements. The syndrome has probably multiple pathophysiological mechanisms, including abnormal axonal branching after aberrant axonal regeneration and enhanced facial motoneuronal excitability. Although the syndrome is relieved with local injections of botulinum toxin, fear of such uncomfortable contractions may lead the patients to avoid certain facial movements, with the implications that this behavior might have on their emotional expressions. © 2003 Movement Disorder Society [source]

    Movement disorders offers electronic manuscript submission

    MOVEMENT DISORDERS, Issue 10 2003
    2004, Anthoy E. Lang MD, FRCPC Editors-in-Chief 199
    [source]

    Bilateral striopallidodentate calcinosis: Clinical characteristics of patients seen in a registry

    MOVEMENT DISORDERS, Issue 2 2001
    Bala V. Manyam MD
    Abstract Clinical features in bilateral striopallidodentate calcinosis (BSPDC), popularly referred to as Fahr's disease (five autosomal dominant families and eight sporadic cases, n = 38), recruited through a registry, are reported. Applying uniform criteria, cases reported in the literature (n = 61) were combined for detailed analysis. The mean (± S.D.) age of Registry patients was 43 ± 21 and that of literature was 38 ± 17. In combined data set (n = 99), 67 were symptomatic and 32 were asymptomatic. Of the symptomatic, the incidence among men was higher compared with women (45:22). Movement disorders accounted for 55% of the total symptomatic patients. Of the movement disorders, parkinsonism accounted for 57%, chorea 19%, tremor 8%, dystonia 8%, athetosis 5%, and orofacial dyskinesia 3%. Overlap of signs referable to different areas of central nervous system (CNS) was common. Other neurologic manifestations included: cognitive impairment, cerebellar signs, speech disorder, pyramidal signs, psychiatric features, gait disorders, sensory changes, and pain. We measured the total volume of calcification using an Electronic Planimeter and Coordinate Digitizer. Results suggest a significantly greater amount of calcification in symptomatic patients compared to asymptomatic patients. This study suggests that movement disorders are the most common manifestations of BSPDC, and among movement disorders, parkinsonism outnumber others. © 2001 Movement Disorder Society. [source]

    Dyskinesias and associated psychiatric disorders following streptococcal infections

    CHILD: CARE, HEALTH AND DEVELOPMENT, Issue 6 2004
    Richard Reading
    Dyskinesias and associated psychiatric disorders following streptococcal infections . DaleRC, HeymanI, SurteesRAH, ChurchAJ, GiovannoniG, GoodmanR & NevilleBGR . ( 2004 ) Archives of Disease in Childhood , 89 , 604 , 610 . Background The classical extrapyramidal movement disorder following , haemolytic streptococcus (BHS) infection is Sydenham's chorea (SC). Recently, other post-streptococcal movement disorders have been described, including motor tics and dystonia. Associated emotional and behavioural alteration is characteristic. Aims To describe experience of post-streptococcal dyskinesias and associated comorbid psychiatric features presenting to a tertiary referral centre 1999,2002. Methods In all patients, dyskinetic movement disorders followed BHS pharyngeal infection. BHS infection was defined by pharyngeal culture of the organism, or paired streptococcal serology. Movement disorders were classified according to international criteria, and validated by experienced child neurologists. Psychiatric complications were defined using ICD-10 criteria using a validated psychiatric interview. Results In the 40 patients, the following dyskinetic movement disorders were present: chorea (n = 20), motor tics (n = 16), dystonia (n = 5), tremor (n = 3), stereotypies (n = 2), opsoclonus (n = 2) and myoclonus (n = 1). Sixty-five per cent of, the, chorea, patients, were, female,, whereas, 69% of the tic patients were male. ICD-10 psychiatric diagnoses were made in 62.5%. Using the same psychiatric instrument, only 8.9% of UK children would be expected to have an ICD-10 psychiatric diagnosis. Emotional disorders occurred in 47.5%, including obsessive-compulsive disorder (27.5%), generalized anxiety (25%) and depressive episode (17.5%). Additional psychiatric morbidity included conduct disorders (27.5%) and hyperkinetic disorders (15%). Psychiatric, movement and post-streptococcal autoimmune disorders were commonly observed in family members. At a mean follow-up of 2.7 years, 72.5% had continuing movement and psychiatric disorders. Conclusion Post-streptococcal dyskinesias occur with significant and disabling psychiatric comorbidity and are potential autoimmune models of common ,idiopathic' movement and psychiatric disorders in children. Multiple factors may be involved in disease expression including genetic predisposition, developmental status and the patient's sex. [source]

    Acculturation is associated with the prevalence of tardive dyskinesia and akathisia in community-treated patients with schizophrenia

    ACTA PSYCHIATRICA SCANDINAVICA, Issue 6 2008
    S. Sundram
    Objective:, Ethnicity is a risk factor for tardive dyskinesia (TD) and other antipsychotic drug-induced movement disorders (ADIMD). It is unclear whether this association is mediated through genetic, environmental or cultural factors individually or in combination. This pilot study aimed to explore this interaction by determining if acculturation in migrant groups contributed to the prevalence of ADIMD. Method:, Culturally diverse but relatively genetically homogeneous (white Caucasian) patients with schizophrenia (n = 40) treated at a single site were assessed for the presence of ADIMD and level of acculturation. Results:, Higher levels of acculturation correlated with an increased prevalence of TD and akathisia but not Parkinsonism. The level of acculturation significantly predicted TD. Conclusion:, This study identifies for the first time that acculturation significantly contributes to the prevalence of TD and akathisia but not Parkinsonism in culturally diverse migrant populations and must be accounted for when explaining ethnic variation in rates of ADIMD. [source]

    Motor stereotypies in children with autism and other developmental disorders

    DEVELOPMENTAL MEDICINE & CHILD NEUROLOGY, Issue 1 2009
    SYLVIE GOLDMAN PHD
    The purpose of the study was to count and characterize the range of stereotypies , repetitive rhythmical, apparently purposeless movements , in developmentally impaired children with and without autism, and to determine whether some types are more prevalent and diagnostically useful in children with autism. We described each motor stereotypy recorded during 15 minutes of archived videos of standardized play sessions in 277 children (209 males, 68 females; mean age 4y 6mo [SD 1y 5mo], range 2y 11mo,8y 1mo), 129 with autistic disorder (DSM-III-R), and 148 cognitively-matched non-autistic developmentally disordered (NADD) comparison children divided into developmental language disorder and non-autism, low IQ (NALIQ) sub-groups. The parts of the body involved and characteristics of all stereotypies were scored blind to diagnosis. More children with autism had stereotypies than the NADD comparison children. Autism and, to a lesser degree, nonverbal IQ (NVIQ) <80, especially in females contributed independently to the occurrence, number, and variety of stereotypies, with non-autistic children without cognitive impairment having the least number of stereotypies and children with autism and low NVIQ the most. Autism contributed independently to gait and hand/finger stereotypies and NVIQ <80 to head/trunk stereotypies. Atypical gazing at fingers and objects was rare but virtually limited to autism. Stereotypies are environmentally modulated movement disorders, some highly suggestive, but not pathognomonic, of autism. Their underlying brain basis and genetic correlates need investigation. [source]