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Laryngeal Atresia (laryngeal + atresia)
Selected AbstractsCongenital lung malformations in the second trimester: Prenatal ultrasound diagnosis and pathologic findingsJOURNAL OF CLINICAL ULTRASOUND, Issue 5 2007Ágnes Harmath MD Abstract Purpose. To correlate prenatal sonographic diagnosis of cystic lung malformations with fetopathologic findings after termination of pregnancy. Methods. We retrospectively analyzed the data of 16 terminated cases in which a cystic lung lesion was diagnosed pre- or postnatally. Results. On average, prenatal diagnosis was established on the 21st gestational week (range, 19,26 weeks). The cause of termination was severe polyhydramnios in 4 cases, nonimmune fetal hydrops in 4 cases, other congenital malformation in 5 cases (renal malformation, 2 cases; congenital diaphragmatic hernia, 3 cases), and obstetrical conditions (intrauterine death, placental abruption, spontaneous abortion) in 3 cases. In 11 cases, congenital cystic adenomatoid malformation (CCAM) was the presumptive prenatal diagnosis. Autopsy confirmed the prenatal diagnosis in 6 of them, while in the other 5 cases, an enteric cyst, a laryngeal atresia, an unidentified tumor, a pulmonary hypoplasia, and an extralobar pulmonary sequestration were found on histologic examination. On the other hand, the autopsy revealed CCAM in those 5 cases in which other malformations were suggested prenatally. Conclusion. The prenatal sonographic diagnosis of CCAM is difficult. Our cases emphasize the important role of fetopathology even today in the verification of prenatal diagnosis based on sonographic examinations. © 2007 Wiley Periodicals, Inc. J Clin Ultrasound, 2007 [source] Severe epiglottic prolapse and the obscured larynx at intubationPEDIATRIC ANESTHESIA, Issue 6 2002F.M. Vaz FRCS BSC (HONS) Summary Laryngomalacia is the most common congenital anomaly of the larynx and usually involves prolapse of the arytenoids, so-called `posterior laryngomalacia'. Most cases resolve with growth of the child and maturation of the larynx, although, rarely, significant airway obstruction can be present. Severe laryngomalacia preventing intubation is rarely encountered. We report a case of a difficult emergency intubation secondary to `anterior laryngomalacia' in a 4-month-old boy in whom the epiglottis prolapsed posteriorly and became trapped in the laryngeal introitus. The child was referred with a diagnosis of laryngeal atresia on the basis of the intubating laryngoscopic view of no apparent epiglottis or laryngeal inlet. The child was transferred with a tube in the oesophagus that kept the child oxygenated. At the time, oxygenation was felt to be due to a coexisting tracheo-oesophageal fistula, although this was eventually found not to be the case. [source] Prenatal diagnosis of laryngeal atresiaPRENATAL DIAGNOSIS, Issue 4 2003L. Öndero Abstract Prenatal diagnosis of congenital atresia of the larynx is difficult but is possible by the findings of increased lung echogenicity and size coexisting with fetal ascites in ultrasonography. Sonographic findings may not always be typical. We report on a case of congenital laryngeal atresia diagnosed prenatally by the findings of fetal hydrops and hyperechogenic lungs. Our case presented with oligohydramnios. We also review syndromes that demonstrate laryngeal anomalies. Copyright © 2003 John Wiley & Sons, Ltd. [source] Congenital laryngeal atresia: two autopsy cases, one describing the use of computed tomographyAPMIS, Issue 5 2000Shigeyuki Asano Two autopsy cases of congenital laryngeal atresia (CLA) were studied. In the first case (a boy delivered at 37 weeks of gestation) the subglottic lumen was almost totally occupied by a dome shaped cricoid cartilage with a pharyngotracheal duct (PTD). No tracheoesophageal fistula (TEF) was found. In the second case (a girl born at 41 weeks of gestation) histology showed that the subglottic lumen was almost occluded by an epithelial lamina and a dome-shaped cricoid cartilage. Again no TEF was found. Both cases had PTD and showed normal development of the lungs. These deformities coincided with Smith & Bain's type II subglottic atresia. The anatomy of the lesion was confirmed by postmortem three-dimensional reconstruction CT, the use of which has not previously been reported. [source] | ||||||||||